Navegando por Palavras-chave "intravascular ultrasound"

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    Pregnancy-associated spontaneous coronary artery dissection: insights from a case series of 13 patients
    (Oxford Univ Press, 2017) Cade, Jamil Ribeiro [UNIFESP]; Szarf, Gilberto [UNIFESP]; Siqueira, Maria Eduarda Menezes de [UNIFESP]; Chaves, Aurea; Andrea, Julio C. M.; Figueira, Helio R.; Gomes Junior, Manuel Pereira Marques [UNIFESP]; Freitas, Barbara P.; Medeiros, Juliana Filgueiras [UNIFESP]; dos Santos, Marcio Ricardo; Fiorotto, Walter B.; Daige, Augusto; Goncalves, Rosaly; Cantarelli, Marcelo; Alves, Claudia Maria Rodrigues [UNIFESP]; Echenique, Leandro [UNIFESP]; Fabio, S. de Brito; Perin, Marco A.; Born, Daniel [UNIFESP]; Hecht, Harvey; Caixeta, Adriano [UNIFESP]
    Aims We sought to present a series of 13 pregnancy-associated spontaneous coronary artery dissection (P-SCAD), their angiographic and multimodal imaging findings, acute phase treatment, and outcomes. Methods and results Between 2005 and 2015, 13 cases of P-SCAD were collected from a database of 11 tertiary hospitals. The mean age was 33.8 3.7 years
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    Tissue characterization and phenotype classification in patients presenting with acute myocardial infarction: Insights from the iWonder study
    (Wiley, 2017) Souza, Cristiano F. [UNIFESP]; Maehara, Akiko; Mintz, Gary S.; Matsumura, Mitsuaki; Alves, Claudia M. R. [UNIFESP]; Carvalho, Antonio Carlos [UNIFESP]; Caixeta, Adriano [UNIFESP]
    ObjectivesWe sought to assess a new modality of radiofrequency intravascular ultrasound (IVUS) called iMAP-IVUS (Boston Scientific, Santa Clara, California) during the evaluation of patients presenting with high-risk acute coronary syndromes. BackgroundThere are limited data on plaque tissue characterization and phenotype classification using iMAP-IVUS. MethodsIn the iWonder study patients presenting with ST-elevation myocardial infarction (STEMI) or non-STEMI underwent three-vessel grayscale IVUS and iMAP-IVUS tissue characterization prior to percutaneous intervention. In total 385 lesions from 100 patients were divided into culprit (n=100) and nonculprit (n=285) lesions. Lesion phenotype was classified as (i) thin-cap fibroatheroma (iMAP-derived TCFA); (ii) thick-cap fibroatheroma; (iii) pathological intimal thickening; (iv) fibrotic plaque; and (v) fibrocalcific plaque. ResultsCulprit lesions had smaller minimum lumen cross-sectional area (MLA) with greater plaque burden compared to non-culprit lesions. Volumetric analysis showed that culprit lesions had longer length and larger vessel and plaque volumes compared to non-culprit lesions. iMAP-IVUS revealed that culprit lesions presented more NC and fibrofatty volume, both at lesion level and at the MLA site (all P<0.001). Any fibroatheroma was more frequently identified in culprit lesions compared with non-culprit lesions (93% vs. 78.9%, P=0.001), anywhere within the lesion 19.0%, P<0.001) as well as at the MLA site (18.0% vs. 9.5%, P=0.07). ConclusionsThree-vessel radiofrequency iMAP-IVUS demonstrated a greater plaque burden and higher prevalence of any fibroatheroma as well as iMAP-derived TCFAs in culprit versus non-culprit lesions in patients presenting with STEMI or non-STEMI undergoing percutaneous coronary intervention. (c) 2017 Wiley Periodicals, Inc.