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dc.contributor.authorReutrakul, Sirimon
dc.contributor.authorThakkinstian, Ammarin
dc.contributor.authorAnothaisintawee, Thunyarat
dc.contributor.authorChontong, Sasipas
dc.contributor.authorBorel, Anne-Laure
dc.contributor.authorPerfect, Michelle M.
dc.contributor.authorPorto Silva Janovsky, Carolina Castro [UNIFESP]
dc.contributor.authorKessler, Romain
dc.contributor.authorSchultes, Bernd
dc.contributor.authorHarsch, Igor Alexander
dc.contributor.authorvan Dijk, Marieke
dc.contributor.authorBouhassira, Didier
dc.contributor.authorMatejko, Bartlomiej
dc.contributor.authorLipton, Rebecca B.
dc.contributor.authorSuwannalai, Parawee
dc.contributor.authorChirakalwasan, Naricha
dc.contributor.authorSchober, Anne-Katrin
dc.contributor.authorKnutson, Kristen L.
dc.identifier.citationSleep Medicine. Amsterdam, v. 23, p. 26-45, 2016.
dc.description.abstractObjectives: The association between inadequate sleep and type 2 diabetes has garnered much attention, but little is known about sleep and type 1 diabetes (T1D). Our objectives were to conduct a systematic review and meta-analysis comparing sleep in persons with and without T1D, and to explore relationships between sleep and glycemic control in T1D. Methods: Studies were identified from Medline and Scopus. Studies reporting measures of sleep in T1D patients and controls, and/or associations between sleep and glycemic control, were selected. Results: A total of 22 studies were eligible for the meta-analysis. Children with T1D had shorter sleep duration (mean difference [MD] = -26.4 minutesen
dc.description.abstract95% confidence interval [CI] = -35.4, -17.7) than controls. Adults with T1D reported poorer sleep quality (MD in standardized sleep quality score = 0.51en
dc.description.abstract95% CI = 033, 0.70), with higher scores reflecting worse sleep quality) than controls, but there was no difference in self-reported sleep duration. Adults with TID who reported sleeping >6 hours had lower hemoglobin A1c (HbA1c) levels than those sleeping <= hours (MD = -0.24%en
dc.description.abstract95% CI = -0.47, -0.02), and participants reporting good sleep quality had lower HbA1c than those with poor sleep quality (MD = -0.19%en
dc.description.abstract95% CI = -0.30,-0.08). The estimated prevalence of obstructive sleep apnea (OSA) in adults with TID was 51.9% (95% CI = 31.2, 72.6). Patients with moderate-to severe OSA had a trend toward higher HbA1c (MD = 0.39%, 95% CI = -0.08, 0.87). Conclusion: T1D was associated with poorer sleep and high prevalence of OSA. Poor sleep quality, shorter sleep duration, and OSA were associated with suboptimal glycemic control in T1D patients. (C) 2016 The Author(s). Published by Elsevier B.V. This is an open access article under the CC BY-NC-ND license.en
dc.description.sponsorshipFather's Day Council
dc.description.sponsorshipUniversity of Arizona Foundation Faculty Small Grants Program
dc.description.sponsorshipNational Institutes of Health (NIH)
dc.description.sponsorshipAmerican Diabetes Association
dc.description.sponsorshipOrder of the Amaranth
dc.publisherElsevier Science Bv
dc.relation.ispartofSleep Medicine
dc.rightsAcesso restrito
dc.subjectSleep qualityen
dc.subjectSleep durationen
dc.subjectObstructive sleep apneaen
dc.subjectType 1 diabetesen
dc.subjectGlycemic controlen
dc.titleSleep characteristics in type 1 diabetes and associations with glycemic control: systematic review and meta-analysisen
dc.description.affiliationMahidol Univ, Ramathibodi Hosp, Fac Med, Div Endocrinol & Metab, Rama 6 Rd, Bangkok 10400, Thailand
dc.description.affiliationMahidol Univ, Fac Ramathibodi Hosp, Sect Clin Epidemiol & Biostat, Bangkok, Thailand
dc.description.affiliationMahidol Univ, Ramathibodi Hosp, Dept Family Med, Bangkok, Thailand
dc.description.affiliationGrenoble Alpes Univ, INSERM, U1042, Hypoxia Pathophysiol HP2 Lab, Grenoble, France
dc.description.affiliationUniv Hosp, Dept Endocrinol, Grenoble, France
dc.description.affiliationUniv Arizona, Dept Disabil & Psychoeduc Studies, Tucson, AZ USA
dc.description.affiliationUniv Fed Sao Paulo, Dept Med, Endocrinol & Diabet Div, Sao Paulo, SP, Brazil
dc.description.affiliationHop Univ Strasbourg, Dept Pneumol, Strasbourg, France
dc.description.affiliationeSwiss Med & Surg Ctr, St Gallen, Switzerland
dc.description.affiliationThuringia Clin Saalfeld Georgius Agr, Dept Internal Med 2, Saalfeld Saale, Germany
dc.description.affiliationLeiden Univ, Med Ctr, Dept Endocrinol & Metab Dis, Leiden, Netherlands
dc.description.affiliationCHU Ambroise Pare, AP HP, INSERM, U987,Ctr Evaluat & Traitement Douleur, Boulogne, France
dc.description.affiliationUniv Versailles St Quentin, Versailles, France
dc.description.affiliationJagiellonian Univ, Coll Med, Dept Metab Dis, Krakow, Poland
dc.description.affiliationUniv Chicago, Inst Endocrine Discovery & Clin Care, Chicago, IL USA
dc.description.affiliationUniv Chicago, Dept Hlth Studies, Chicago, IL USA
dc.description.affiliationMahidol Univ, Ramathibodi Hosp, Fac Med, Div Allergy Immunol & Rheumatol, Bangkok, Thailand
dc.description.affiliationChulalongkorn Univ, Fac Med, Dept Med, Div Pulm & Crit Care Med, Bangkok, Thailand
dc.description.affiliationKing Chulalongkom Mem Hosp, Thai Red Cross Soc, Excellence Ctr Sleep Disorders, Bangkok, Thailand
dc.description.affiliationReg Hosp Feldbach Furstenfeld, Dept Internal Med, Furstenfeld, Austria
dc.description.affiliationUniv Chicago, Dept Med, Sect Pulm & Crit Care, 5841 S Maryland Ave, Chicago, IL 60637 USA
dc.description.affiliationUnifespUniv Fed Sao Paulo, Dept Med, Endocrinol & Diabet Div, Sao Paulo, SP, Brazil
dc.description.sponsorshipIDNational Institutes of Health (NIH): HL62373
dc.description.sponsorshipIDAmerican Diabetes Association: 7-13-CE-32
dc.description.sourceWeb of Science

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